Phytate-phosphorus and phytase on performance, bone characteristics, tissue and serum mineral concentration on broilers.

Male broiler chickens (384), Cobb 500, were housed in metabolic cages to assess the efficacy of phytase in diets with low and high phytate-phosphorus on the performance, bone physical characteristics, tissue and serum mineral deposits. Birds were distributed in four treatments with a 2 x 2 factorial arrangement in a completely randomized block design. Experimental diets based on maize-soybean meal were T1 - diet low phytate-phosphorus; T2 - diet low phytate-phosphorus and phytase (500 FTU/kg); T3 - diet high phytate-phosphorus; T4 - diet high phytate-phosphorus and phytase (500 FTU/kg). Feed intake, body weight, weight gain and feed conversion ratio were assessed. Two left tibias per experimental unit were analyzed for physical characteristics and mineral concentration; a section of skinless breast muscle and blood were collected to measure the concentration of calcium, phosphorus and sodium. Results showed interaction between bone stiffness and serum calcium. The inclusion of phytase in diets with low and high phytate-phosphorus did not alter performance, bone resistance and flexibility, mineral deposits in the tibia and breast muscle, but increased bone stiffness after 22 days of age. It also provided a higher serum calcium rate in broilers fed diets with low phytate-phosphorus up to 32 days of age.

Effect of pelleting and different feeding programs on growth performance, carcass yield, and nutrient digestibility in broiler chickens.

This experiment was conducted to study the effect of different feeding programs and pelleting on performance, nutrient digestibility, ileal digestible energy (IDE); and carcass yield of broilers from 21 to 35 d of age. In total, 768 male broilers were distributed according to a completely randomized design with 6 treatments and 8 replicates of 16 birds each. The treatments were mash and pelleted diets provided ad libitum, or pelleted and supplied at the same rate (100%) or restricted at 95, 90, and 85% (P100, P95, P90, and P85) of the amount consumed by the birds fed mash diet ad libitum. When supplied ad libitum, the pelleted diet had the highest feed intake and weight gain (WG), better feed conversion ratio (FCR), better feed conversion adjusted for 2.3 kg (AdjFCR, P < 0.001) and caloric conversion (P < 0.001); and higher amount of abdominal fat (P < 0.001) when compared to the control (mash ad libitum). However, there were no effects on nutrient digestibility (P > 0.05). When the pelleted feed was provided in the same amount as in the control group, there were no differences in any of the evaluated parameters (P > 0.05). Limiting pelleted diet to 95, 90, and 85% of free choice mash diet resulted in lower WG (P < 0.001). P90 and P95 treatments resulted in higher dry matter and crude protein digestibility and IDE in relation to the others (P < 0.001). Carcass yield was reduced (P < 0.05) in the birds fed P85 diet. The regression analysis between P100, P95, P90, and P85 showed a linear reduction in WG when restriction was increased (P < 0.01); however, there was a linear increase in the nutrient digestibility (P < 0.001). It is concluded that pelleting improves broiler performance, but these results depend on feed intake. The higher intake provided by pelleting can increase the amount of abdominal fat. Feed intake reduction can result in lower performance and lower carcass and cuts yield in broilers.

The effects of ochratoxin/aluminosilicate interaction on the tissues and humoral immune response of broilers.

This study aimed to evaluate the effect of dietary ochratoxin, in the presence or absence of aluminosilicate, on the histology of the bursa of Fabricius, liver and kidneys, and on the humoral immune response of broilers vaccinated against Newcastle disease virus. The exposure of birds to 2 p.p.m. ochratoxin, in the presence or absence of aluminosilicate, reduced their humoral immune response and the number of mitotic cells in the bursa. The relative weight of the livers of the birds exposed to this toxin was increased and, microscopically, there was hepatocyte vacuolation and megalocytosis with accompanying hyperplasia of the biliary epithelium. The kidneys showed hypertrophy of the renal proximal tubular epithelium, with thickening of the glomerular basement membrane. Aluminosilicate did not ameliorate the deleterious effects of the ochratoxin.